Hsa?miR?503?5p regulates CTDSPL to accelerate cisplatin resistance and angiogenesis of lung adenocarcinoma cells
Chemical Biology and Drug Design
JUNE 21, 2023
High expression of hsa-miR-503-5p and decreased expression of CTDSPL in LUAD contribute to cisplatin resistance and angiogenesis. Hsa-miR-503-5p negatively regulates CTDSPL, suggesting its potential as a target to overcome cisplatin resistance in LUAD.
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